Seifter J.L. Seifter, Julian cción de vías urinarias. INTRODUCCIÓN; ETIOLOGÍA; MANIFESTACIONES CLÍNICAS Y FISIOPATOLOGÍA; DIAGNÓSTICO Asimismo, la uropatía obstructiva quizá sea resultado de una neoplasia. Existen pocos datos o signos clinicos que puedan orientar al diagnostico de RVU. Este se basa en la frecuencia de los hallazgos de este. Pérdida del funcionamiento normal de la vejiga provocada por alteración de la inervación vesical que origina un trastorno en el fenómeno de.

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This phenomenon could be mediated by the uropatiw of angiotensin II and tromboxane by the obstructed nephrons. The obstruction of the urinary flow can take place inside the renal tubules as well as in any other segment of the urinary tract renal pelvis, ureter, bladder and urethra.

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Hospital Italiano de Buenos Aires. It is worth mentioning that hydronephrosis is the expansion of the pelvis and renal calyces proximal to the obstruction point; and that expansion is not always synonym of obstruction, since there are non-obstructive types of expansion known as ectasias 1 Tabla 1.

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Uropatia obstructiva by Paula Tamara Mohamad on Prezi

Pop-up div Successfully Displayed This div only appears when the trigger link is hovered over. What happens during a complete and bilateral uro-obstruction is that the hydrostatic pressure of the Bowman capsule increases greatly, and it can even override the net ultrafiltration pressure and lead to obstructive renal failure.

Partial outlet obstruction in rabbits: On the other hand, such pressure is transmitted to the tubular sectors proximal to the obstruction causing a reduction of the glomerular filtration since it counteracts to the glomerular filtration net uropatiw.

Can Urol Assoc J.

Obstrucción de vías urinarias | Harrison. Principios de Medicina Interna, 18e | McGraw-Hill Medical

Usually glomerular hydrostatic pressure gisiopatologia largely predominant, on whom the net ultrafiltration pressure depends almost completely. Arch Ital Urol Ostructiva. Published, October 8, After the resolution of a bilateral obstruction or a obstrructiva one in a patient with only one kidney, it is normal to find elevated serum levels of atrial factors, tubular resistance to vasopressin reduction of the expression of aquaporin 2 channels in the collecting tubules and compromise of the medullar tonicitydecrease in the tubular reabsorption capacity of sodium and uropagia and presence of a free urinary tract, so the osmotic diuretic effect of the not reabsorbed urea and sodium starts to act, which increment diuresis finally leading to potassium, calcium, magnesium and phosphorus expoliation, which puts the patient at risk of having severe hydroelectrolytic depletion if these losses are not adequately monitored and treated.


Comment of the reviewer Jesus Garrido MD. About MyAccess If your institution subscribes to this resource, and you don’t have a MyAccess Profile, please contact your library’s reference desk for information on how to gain access to this resource from off-campus.

Role of angiotensin II in chronic ureteral obstruction. Transurethral prostate resection in patients with hypocontractile detrusor–what is the predictive value of ultrastructural detrusor changes? You can also find results for a single author or contributor. View Table Favorite Table Download. Sign in via Shibboleth. A later resolution can mean partial or nule recovery, depending on the evolution time of the obstruction, the age of the patient and the degree of damage to the renal function previous to the obstruction.

The latter can be subdivided into those which have intrinsic and extrinsic causes to the urinary tract Increase in uropaatia wall thickness indicates bladder outlet obstruction BOO in men.


Sometimes there is a rapture of the renal calices with the subsequent formation of urinomas. In general, this condition known as post desobstructive poliuria, usually self-constraints in three days and does not extend for longer than a week. In conclusion, since obstructive nephropathy is a potentially reversible cause of renal dysfunction, it should always be taken into account among the differential diagnosis of renal failure inducing mechanisms. There are many renal dysfunction inducing mechanisms involved in this entity: Adv Exp Med Biol.


Nevertheless, if the obstruction is sustained in time, it leads to intrarenal vasoconstriction with the subsequent reduction in the glomerular blood flow. Search Advanced search allows to you precisely focus your query.

Clinical Sports Medicine Collection. In the case of intratubular obstructions uric acid, pigments, etc. Obstruction-induced alterations within the urinary bladder and their role in the pathophysiology of lower urinary tract symptomatology.

Accessed December 31, Principios de Medicina Interna, 18e. Likewise, the urinary obstruction can lead to a dysfunction of the distal nephron sectors resistance to aldosterone and vasopresinmaking it difficult for the local secretion of potassium and protons, as well as reducing the water reabsorption, thus facilitating the development of hyperkalemia, hyperchloremic metabolic acidosis and nephrogenic diabetes insipidus, obstryctiva. In Schrier R Ed.

Servicio de ayuda de la uropqtia. View All Subscription Options. Am J Physiol Renal Physiol.

Am J Physiol Renal Physiol ; The aging kidney in health and disease. Obstructive uropathy is a mechanism of renal insufficiency, which since it is relatively simple to solve, should always be taken into consideration as one of the differential diagnosis of renal failure.

Factors determining the ohstructiva of residual urine in men with bladder outlet obstruction: It is worth mentioning that hydronephrosis is the expansion of the pelvis and renal calyces proximal to the obstruction point; and that expansion is not always synonym of obstruction, since there are non-obstructive types of expansion known as ectasias 1 Tabla 1 Physiopatology Curr Opin Nephrol Hypertens ;